The True Secret To Slower Aging
When we think about turning back the hands of time, one may recall the story of the Spanish explorer, Ponce de Leon and his search for the Fountain of Youth. De Leon never did find The Fountain. He is not alone, as millions of people, to this very day, are actively searching for just that – staying youthful, as long as possible. I am excited to share with you a novel nutrient I found, called Mitopure, which is unlocking the pathways within our body that enable renewal and revitalization. It is my belief that these pathways hold the promise of slowing, and in many cases, reverse aging.
Mitopure is giving my readers ONLY a 10% discount. Use this discount offer, LORI10 for 10% off.
Nobody said getting older was a breeze. We easily recognize that the older we get the slower, heavier, and lazier we feel. In fact, beginning around our 30th birthday, the body begins to slow down and that includes the reduction of muscle mass. In fact, muscle mass decreases approximately 3–8% per decade after the age of 30 and this rate of loss is exponential after the age of 60. This is one of the most striking effects of aging – the loss of muscle mass, strength, and function. There is a direct correlation that exists between how we feel: the slowing of our metabolism, loss of mental sharpness and mobility with the loss of muscle mass. This is why muscle is crucial to optimal health. It turns out supporting your muscles, all the way down to the cellular level, is one of the best things you can do to optimize healthful aging.
I became interested in the search of optimizing my cellular health years ago and have been actively involved in achieving slower aging and a better quality, more youthful life both personally and for my clients professionally.
In my journey, I became interested in how crucial the mitochondria are, not just to life itself, but also to the process of supporting muscular health – a key to slowing and reversing cellular aging. This is where Mitopure makes all the difference; but first, let’s cover some of the basics.
Mitochondria
What are Mitochondria?
Mitochondria are tiny organelles that are at the heart of muscle and aging. They are tiny factories housed within our cells that take the foods we eat and the oxygen we breathe and convert them into energy called adenosine triphosphate or ATP. This energy is crucial in that it keeps us alive. Without robust mitochondria, cells cannot do what they’re capable of and why we need them to stay healthy.
To achieve higher levels of performance we must optimize our mitochondria, the powerhouse of our cells.
Mitochondria and Aging
Mitochondria generate the ATP energy that fuels everything your body does from powering your heart beat, to contracting muscle, and even powering your brain to process information.
Have you ever noticed how children have an abundance of energy? This is not just because they are younger, this is due, from a scientific standpoint, to the abundance and health of their mitochondria. Unfortunately, over time our mitochondria become less and less capable.
What triggers poor mitochondrial health? Cell damage is caused my many sources but one that we control is the food we eat. Science is revealing that certain things we eat such as sugar, processed foods, and industrial processed cooking oils take a heavy toll on our cellular health. Excess food intake, smoking, and our modern, sedentary lifestyle are also to blame for the multitude of health issues being linked to mitochondrial damage.
Poor functioning mitochondria lead to oxidative stress, inflammation, low energy, weight gain and obesity, metabolic syndrome, memory loss, cognitive decline and rapid aging. Why? Because the mitochondria are not producing adequate ATP.
This leads us to the very important question: If we have been harming our mitochondria, is it too late to repair the damage? The answer is a resounding NO – it is never too late.
NBC’s Nutritionist Joy Bauer
Your Body’s Natural Defense Against Aging
At a cellular level, our body is constantly destroying old cells and replacing them with new ones. This cellular housecleaning is called autophagy. The word “autophagy” comes from the Greek: “auto” or self and “phagy” or eat. That is exactly what the body is doing at the cellular level, breaking down old worn-out parts and recycling them into new parts.
When autophagy happens within the mitochondria it is called mitophagy. If sick or impaired mitochondria are hanging out, mitophagy ensures that these unhealthy mitochondria are removed – nature’s perfect way to protect us from cellular decline. Research has shown, however, that our sedentary lifestyles and access to abundant food have short-circuited the natural housecleaning. The result is an increase in dysfunctional mitochondria and a reduction in cellular energy which contributes to our feeling of aging.
Why is Muscle Vital to Slower Aging?
Muscle loss, frailty, and lack of strength are hallmarks of aging. Lack of muscle as we age compromises our metabolism, bones and mental sharpness. Essentially having enough muscle mass as we age affects every area of health, including longevity.
Mitochondria, those powerhouses we have been talking about, are present in cells all over our bodies from our brains to our hearts, but they are most abundant in muscle tissue. Makes sense, muscle needs tons of energy to function optimally. You can imagine when the mitochondria that generate muscular energy become damaged and accumulate it leads to dysfunction throughout the body. Restoring our body’s natural renewal process of mitophagy is the key.
Urolithin A, the A should stand for Amazing
If only there were a way to trigger mitophagy, the body’s natural renewal process we just discussed. During my research, I stumbled upon the pomegranate, valued since ancient times for its longevity supporting properties. Today, research is revealing the pomegranates role in our quest for longevity, and the findings lead directly to a compound called Urolithin A.
Urolithin A is generated by our gut microflora as a natural food metabolite of ellagitannins, a class of compounds found in the pomegranate and other fruits, nuts and tea. Let me say that again in plain language so you don’t miss this critical part. When you eat pomegranates your digestion process breaks the fruit down and your body makes Urolithin A. Urolithin A is not in pomegranates or any food. It is made by your body. Amazing.
Not Everyone Can Make Urolithin A
Research suggests that most of us can’t produce Urolithin A. Perhaps it is due to our Western diet or our modern lifestyles, but most of us (research has found as many as 60-70%) don’t have the right gut microbiome to produce Urolithin A. For us, even drinking several glasses of pomegranate juice simply won’t work. In fact drinking juice is simply drinking liquid sugar that may promote inflammation, weight gain and other metabolic conditions.
For the lucky few who can produce Urolithin A, there is another consideration. It is unclear how much ellagitannin rich fruit and nuts one would have to eat to make a therapeutic amount of Urolithin A, it is possible this is different for each of us.
Until recently, that is where the story would have ended, just like Ponce De Leon, fruitless. Fortunately, Urolithin A and its remarkable ability to trigger mitophagy — cellular renewal and revitalization — is now available to you.
Mitopure, Urolithin A for Every Body
The product of 10 years of groundbreaking research by a world class scientific team, Mitopure unlocks all of the benefits of Urolithin A and can be used by anyone. I imagine you have questions, so let’s clear up some of the likely ones:
Does it work even if my body doesn’t make Urolithin A?” Yes!
Will I get a therapeutic amount of Urolithin A so I can ensure I trigger my body’s natural housecleaning process?” Yes. Mitopure delivers 6x more Urolithin A than 8 ounces (240 ml) of pomegranate juice. Regardless of whether you are a natural Urolithin A producer or not, you get the same effective nutrient boost.
Is it safe? The US Food and Drug Administration (FDA) thinks so. They classified Mitopure as “Generally Recognized As Safe” (GRAS), their highest safety classification.
Where do I get Mitopure? Mitopure is at the heart of all Time-Line Nutrition’s products.
Mitopure is giving my readers a 10% discount. Use this discount offer, LORI10 for 10% off.
The True Secret to Slower Aging
Why do children have so much bountiful, unstoppable energy? In part, it is because they have an abundance of healthy mitochondria and growing muscles. Mitopure is clinically proven to improve muscle strength and function and revitalize mitochondria. By unlocking the natural power of Urolithin A and making it available to everyone, Mitopure has introduced a new class of nutrition that wasn’t widely available just a few short years ago. Change your health starting today!
*Lori Shemek, PhD, CNC is a Nutritional Consultant for Mitopure.
References
Ryu, D. et al. Urolithin A induces mitophagy and prolongs lifespan in C. elegans and increases muscle function in rodents. Nat. Med. 22, 879–888 (2016).
Ryu, D. et al. Urolithin A induces mitophagy and prolongs lifespan in C. elegans and increases muscle function in rodents. Nat. Med. 22, 879–888 (2016).
Garcia-Villalba R. et al. Gastrointestinal Simulation Model TWIN-SHIME Shows Differences between Human Urolithin-Metabotypes in Gut Microbiota Composition, Pomegranate Polyphenol Metabolism, and Transport along the Intestinal Tract.
Ryu, D. et al. Urolithin A induces mitophagy and prolongs lifespan in C. elegans and increases muscle function in rodents. Nat. Med. 22, 879–888 (2016).
Heilman, J. et al. Safety assessment of Urolithin A, a metabolite produced by the human gut microbiota upon dietary intake of plant derived ellagitannins and ellagic acid. Food Chem Toxicol. 2017 Oct;108(Pt A):289-297. Epub 2017 Jul 27.
Singh, A. et al. Orally administered urolithin a is safe and modulates muscle and mitochondrial biomarkers in elderly. Innovation in Aging 1, 1223–1224 (2017).
The mitophagy activator Urolithin A is safe and induces a molecular signature of improved mitochondrial and cellular health in humans” (doi: 10.1038/s42255-019-0073-4).